<?xml version="1.0" encoding="UTF-8"?><rss version="2.0" xmlns:content="http://purl.org/rss/1.0/modules/content/" xmlns:dc="http://purl.org/dc/elements/1.1/" xmlns:atom="http://www.w3.org/2005/Atom" xmlns:sy="http://purl.org/rss/1.0/modules/syndication/" > <channel><title>Comments on: Extinction Fears of the Red-Headed Homo Sapien</title> <atom:link href="http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/feed/" rel="self" type="application/rss+xml" /><link>http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/</link> <description>Topics from multidimensional biopsychosocial perspectives.</description> <lastBuildDate>Thu, 18 Mar 2010 21:46:47 +0000</lastBuildDate> <generator>http://wordpress.org/?v=2.9.2</generator> <sy:updatePeriod>hourly</sy:updatePeriod> <sy:updateFrequency>1</sy:updateFrequency> <item><title>By: mark</title><link>http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-155464</link> <dc:creator>mark</dc:creator> <pubDate>Sat, 26 Apr 2008 01:07:16 +0000</pubDate> <guid isPermaLink="false">http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-155464</guid> <description>Measurement of the genomic rate of deleterious mutations (generally abbreviated as &#039;U&#039;) in both species. By looking at the generalization P and the nit-picking V&#039; and s&#039; constant can be omitted in favor of non-synonymous (dN) and synonymous (dS)  of near-native paradoxical observed retention mechanism conferred by the U allele or association of standard chr.1 thru U and X-Y genotyping techniques regarding the causal role of common human DNA variation in L5 human populations is no reason for a L3 inference. Based upon obvious morphological features during three consecutive generations. At the mechanical level of manipulating the technical factor for comparing the rodent mutant strain as predicted inheritable to the Human genomic database U [undifferentiated] in the MC1R (melanocortine 1 receptor gene)â€¦ Y2 receptors regulated transcript (CART) in the arcuate nucleus. Anorexic proopiomelanocortin (POMC)conditional knockouts could be sustained 3&#039;-5&#039; by high stability of the 5&#039;end by the 3&#039; end of the same genotyped RNA[?!]... and would be to adopt the antisymmetry to granular cells by so-called brain doping to imprint the induced response: the &lt;a href=&quot;http://www.idi.ntnu.no/~aleks/farting.txt&quot; rel=&quot;nofollow&quot;&gt;latency of containment like syndrome&lt;/a&gt;, here by inference herein B.B. of course.</description> <content:encoded><![CDATA[<p>Measurement of the genomic rate of deleterious mutations (generally abbreviated as &#8216;U&#8217;) in both species. By looking at the generalization P and the nit-picking V&#8217; and s&#8217; constant can be omitted in favor of non-synonymous (dN) and synonymous (dS)  of near-native paradoxical observed retention mechanism conferred by the U allele or association of standard chr.1 thru U and X-Y genotyping techniques regarding the causal role of common human DNA variation in L5 human populations is no reason for a L3 inference. Based upon obvious morphological features during three consecutive generations. At the mechanical level of manipulating the technical factor for comparing the rodent mutant strain as predicted inheritable to the Human genomic database U [undifferentiated] in the MC1R (melanocortine 1 receptor gene)â€¦ Y2 receptors regulated transcript (CART) in the arcuate nucleus. Anorexic proopiomelanocortin (POMC)conditional knockouts could be sustained 3&#8242;-5&#8242; by high stability of the 5&#8242;end by the 3&#8242; end of the same genotyped RNA[?!]&#8230; and would be to adopt the antisymmetry to granular cells by so-called brain doping to imprint the induced response: the <a href="http://www.idi.ntnu.no/~aleks/farting.txt" rel="nofollow">latency of containment like syndrome</a>, here by inference herein B.B. of course.</p> ]]></content:encoded> </item> <item><title>By: Anonymous</title><link>http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-154093</link> <dc:creator>Anonymous</dc:creator> <pubDate>Fri, 25 Apr 2008 02:02:39 +0000</pubDate> <guid isPermaLink="false">http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-154093</guid> <description>This is very offensive!</description> <content:encoded><![CDATA[<p>This is very offensive!</p> ]]></content:encoded> </item> <item><title>By: Matt</title><link>http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-150926</link> <dc:creator>Matt</dc:creator> <pubDate>Tue, 22 Apr 2008 12:41:51 +0000</pubDate> <guid isPermaLink="false">http://brainblogger.com/2008/02/17/extinction-fears-of-the-red-headed-homo-sapien/#comment-150926</guid> <description>Well if what u say is true then the red head&#039;s will live on because the resesive gene has been known to over ride the dominate gene.  When someone has B,r and b is brown and r is red so the r might overwrite the B gene...  Though this is unlikly it has been known to happen in some rare cases.  And also it will live on because the red hair may spring up in a later generation, from the original parents.</description> <content:encoded><![CDATA[<p>Well if what u say is true then the red head&#8217;s will live on because the resesive gene has been known to over ride the dominate gene.  When someone has B,r and b is brown and r is red so the r might overwrite the B gene&#8230;  Though this is unlikly it has been known to happen in some rare cases.  And also it will live on because the red hair may spring up in a later generation, from the original parents.</p> ]]></content:encoded> </item> </channel> </rss>
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